2014年12月12日星期五

Micro-Chinese Medicine therapy for polycystic kidney penetration effect

Micro-Chinese Medicine therapy

Micro-Chinese Medicine Osmotherapy therapy for polycystic kidney penetration effect how? Micro-Chinese Medicine therapy for polycystic kidney disease will relapse penetrate it? There are many patients who do not understand the micro-penetration of traditional Chinese medicine therapy for polycystic kidney disease will produce what kind of effect,

In medicine is a congenital hereditary polycystic kidney disease, kidney essence covered countless sizes ranging from communicating with the outside world is not round cyst capsule containing a liquid, a small invisible, large There may be several centimeters, so called polycystic kidney disease. Expressed as nocturia, lower back pain, high blood pressure. Urinalysis hematuria, a small amount of proteinuria, often slowly progressed to chronic renal failure. 10% of people with kidney stones, 30% of people with polycystic liver. Experienced doctors With B ultrasound, intravenous urography can be confirmed.

The use of micro-penetration of traditional Chinese medicine therapy for adult polycystic kidney disease can be manipulated to achieve the effect, then the micro-penetration of traditional Chinese medicine therapy cure, polycystic kidney disease results so far?

Micro-based therapy penetration through the skin, blood, and the meridian system of the body lower back, straightforward effect on kidney lesions in adult polycystic kidney disease, open and efficient release of Chinese medicine, local drug concentration, bioavailability use, straightforward role in kidney disease organization, kidney lesions have targeted positioning, the complete eradication of immune complexes and necrotic tissue has completely eradicated stronger, fast and long-lasting effect to stop the progression of polycystic kidney disease to chronic renal failure. Moreover, micro-penetration of traditional Chinese medicine therapy for adult polycystic kidney disease also has no pain, short course, quick, and patients with polycystic kidney disease self-treatment at home, and shorten hospital stays, thrift invalid loss.

2014年12月11日星期四

Chinese and Western medicine prevention of chronic renal failure advantage

Kidney Failure is a variety of kidney disease progression to the last common outcome is a progressive development of the disease. Blood purification therapy and kidney transplantation is an important means of treatment of the disease, but not limited to the conditions still popular, and therefore non-dialysis therapy more practical significance. After 30 years of Chinese scholars unremitting efforts, the use of non-dialysis therapy Integrative CRF has made remarkable achievements. Integrative core non-dialysis therapy is to relieve symptoms and protect residual renal function, and delay the progression of delayed dialysis and kidney transplantation have time, greatly enhance the quality of life of life of patients.

1. Kidney Failure  caused by many reasons for the cause of prevention, we must seriously find primary disease, the cause of targeted therapy to prevent further deterioration of renal function. In the primary kidney disease, chronic nephritis occupies first place, followed by chronic pyelonephritis, in secondary renal disease, diabetic nephropathy, hypertensive renal damage is more common. Chinese medicine in the prevention and treatment of primary kidney disease can effectively control its evolved Kidney Failure, and Integrative Medicine Prevention also has important clinical significance in secondary renal disease.

2. Control reversible causes of Kidney Failure is a progressive course of development, but influenced by certain factors, at a certain stage, there may be a sharp deterioration in renal function. Therefore timely dispel these reversible factors, is an important measure to prevent the rapid deterioration of renal function. Prompting a sharp deterioration in renal function main factors (1): ① involving kidney disease recurrence or worsening; ② water and electrolyte disorders; ③ various infections; ④ hypertension, heart failure; ⑤ stress; ⑥ other adverse factor. These reversible factors using conventional Western measures often can achieve satisfactory results. Chinese medicine believes that its reversible factors are (2): ① exogenous wind-heat or cold; ② phlegm; ③ wet; ④ dampness; ⑤ heat; ⑥ stasis; ⑦ wind; ⑧ air dry. Chinese medicine against these evil empirical, in addition to the commonly used oral decoction syndrome, but also the use of such special party, special medicine, internal, external washing, enemas, intravenous administration and other measures, not only a good effect, but also the wonderful specimens wen.

3. Diet plus essential amino acids (EAA) therapy low-protein diet may reduce glomerular blood perfusion, so that the "healthy after renal unit," the filtration rate reduction, slow process of glomerular sclerosis, thereby protecting the remaining kidney function. In order to prevent negative nitrogen balance and unfavorable factors brought low protein diet, present, many advocates using (1): ① low protein diet + EAA therapy; ② low-protein diet + α- keto acid therapy. Diet has always been valued by traditional Chinese medicine, but because of the particularity of CRF, although like all deficiency, but its "gasification inadequate", prone to toxic cloud. Therefore, the "filling in the empty" and can not stay up, tired of fill, fill pure, must fill and with restraint, especially gasification kidney function should be and evil and embrace the situation as a reference, syndrome tonic, so better CRF's compliance with the disease.

The best way to treat chronic renal failure

the best way to treat chronic renal failure

was suffering kidney failure often behave very scared that the disease is incurable, and even they think this is an incurable disease. We do not deny the severity of renal failure, renal failure, but this is considered "incurable" That's some exaggeration, but this is a deep misunderstanding of the kind of renal failure.

If patients with renal failure patients receive formal scientific treatment as early as possible to avoid dialysis, reducing the number of dialysis, dialysis prolonged intervals until they get rid of dialysis are entirely possible. If you control the complications in some patients, the disease is fully able to control the life and quality of life will not be affected substantially. Therefore, patients with renal failure and do not panic, timely and correct treatment is to treat the disease that some gesture.

Currently only treat Kidney Failure, uremia approach is not much. Dialysis and kidney transplant is the most common method of treatment of Kidney Failure, dialysis can replace kidneys excrete metabolic toxins play a role in the patient's excessive accumulation of metabolic waste from the body, patients with high toxin temporarily relieve the state, so that patients in the shortest time out of danger range. But the long-term dialysis treatment will increase the patient's own symptoms, there will be a lot of complications, such as concurrent cardiovascular disease, complicated by infection, anemia, bone disease and so on dialysis.

Kidney transplantation is the treatment of Kidney Failure in one of the most direct, but the kidney transplant surgery on the one hand to the hospital, medical staff are higher, on the other hand, kidney transplants are also facing the right kidney and high double surgery difficulties, the average family simply can not afford to accept. Furthermore, even if doing kidney transplants, patients need to take anti-rejection drugs for life, can be described as a mixed blessing. Related statistics show that dialysis and kidney transplant survival time is essentially similar, can live over a year about 90% of patients can survive for five years of approximately 5% of patients. Thus dialysis and kidney transplant is not the best way to treat chronic renal failure can be seen.

Chronic nephritis can eat carrots


For us, now is a relatively common chronic nephritis kidney disease, there are a lot of people like to eat carrots, then chronic nephritis patients can not eat carrots it? Our experts dedicated to an analysis of the following points.

Nephritis patients often show no obvious back pain, but some patients find nephritis, nephritis waist is sick, so he felt back pain, back pain and kidney stones great as pyelonephritis, back pain, back pain, mostly pyelonephritis side, back pain and kidney stones and a lot of very intense radiation to the inside of the thigh.

Nephritis occurs in patients with edema, edema often first appear in the eyelids, face, scrotum and other places relatively loose lower limb edema, severe edema can also be a small number of people may have ascites. Secondly, increased urinary talk bubble. Increased urinary protein foams generally have the phenomenon, in general, the foam, the more protein. Then say hematuria. Under normal circumstances, urine in the human eye can not see the blood, and the microscope is no red blood cells, if there are gross hematuria urine red blood cells means that there are a lot. However, this does not mean that red is the color of urine hematuria, of course, but it does not necessarily mean nephritis. Also, pay attention lesbian hematuria is not caused by the menstrual period, so to avoid the menstrual period, urine tests.

Radish is very rich in nutrients, contains the body's own production of interferon can induce a variety of trace elements, can enhance immunity, and can inhibit the growth of cancer cells, cancer prevention, cancer is important. Radish mustard oil and dietary fiber can promote gastrointestinal peristalsis, helps to expel waste. Eat carrots can reduce blood fat, soften blood vessels, blood pressure, prevention of coronary heart disease, arteriosclerosis, cholelithiasis and other diseases.

Chronic nephritis also can eat radish. General population can eat carrots. But a weak constitution, spleen and stomach, gastric and duodenal ulcers, chronic gastritis, simple goiter, threatened abortion, uterine prolapse should not eat.

2014年12月10日星期三

Ten taboos chronic kidney disease


A bogey drinking. Drinking alcohol fusel oil and nitrite limb can kidney tissue degeneration and cancer,  chronic kidney disease and therefore should not drink at all, in order to avoid liver and kidney damage.

Second, avoid smoking. Tobacco contains a variety of harmful substances that can damage the liver and kidney function inhibit renal unit repair,  chronic kidney disease must therefore determined to quit smoking.

Three bogey angry. Medicine for Yu Nu liver injury, liver qi stagnation is not stretched, resulting in detoxification, detoxification function was transformed into the kidneys, increase the burden on the kidneys, easy to plot addiction, so angry was the enemy of the kidney.

Four bogey fatigue. Kidney is an important human metabolic organ, chronic kidney disease patients with renal dysfunction, malnutrition, fatigue, weakness and therefore, need more rest.

Five Note anxiety. Kidney disease (especially in patients with uremia) permanently, often people anxious, the cerebral cortex of high tension, for kidney disease (especially female patients) Rehabilitation extremely unfavorable.

Bogey six pessimistic. Nephritis, uremia once they lose confidence in the treatment, the disease is more difficult to control, so the patient to be optimistic, open-minded, and enhance confidence.

Seven avoid indiscriminate use tonic. A balanced diet is essential to maintain good health conditions, such as tonic improper tip the balance will affect their health, and therefore caution tonic.

Eight bogey irregular life. Seven support very sick, so adequate sleep, good nutrition, regular life is crucial for kidney patients, sexual restraint, keep the essence is to maintain and establish awareness of life.

Nine avoid drug abuse. Three-drug, drug for more than a liver and kidney damage, kidney patients must be rational use of drugs under the guidance of specialists, and the early acceptance of the system, formal, scientific treatment.

Ten taboos trying everything. Not believe in the political arena Youyi, lest the disease is not cured, not less money to spend, to delay the disease repentant.

Early symptoms of Chronic Kidney Disease which

early detection of Chronic Kidney Disease
early detection of Chronic Kidney Disease
Early symptoms of Chronic Kidney Disease include: weakness, fatigue, back pain, edema dispensable, edema sites often appear in the eyelids, face and lower limbs, usually mild to moderate edema, rarely seen in patients with Chronic Kidney Disease did not cause uremia pleural effusion, ascites, etc., which, weakness, fatigue, back pain and so are more familiar with, but because the symptoms are more widespread, so in the event of such symptoms, the need to be combined with other related symptoms or laboratory tests confirmed again.

Experts advise: To early detection of Chronic Kidney Disease
Under normal circumstances, the incidence of Chronic Kidney Disease accompanied by a series of symptoms manifested. Early symptoms of Chronic Kidney Disease in various forms, some hidden, some obvious. The following will introduce three kinds of common early symptoms of Chronic Kidney Disease, easy to reference controls. If a similar problem, timely medical diagnosis, to avoid delays in treatment.

One of the early symptoms of Chronic Kidney Disease: eyelid edema
The main reason for early Chronic Kidney Disease is prone to swelling of the eyelids: renal excretion of water damage and regulatory function, so that children increase the body's water and sodium, too much water accumulated in the body loose organization, and eyelid is loose tissue more parts. Eyelid edema is characterized by a clear morning when, after hypoactivity.

Second, the early symptoms of Chronic Kidney Disease: hematuria
Hematuria gross hematuria and microscopic hematuria points. That is visible to the naked eye hematuria hematuria, urinary Secheng wash the meat in water samples, turbidity and red, some children in urine or blood clots in the folder with bloodshot eyes; and microscopic hematuria only observed under a microscope can be found, the number of red blood cells per high power field under more than one. Most children hematuria is the most common symptoms of Chronic Kidney Disease patients. Of course, many reasons hematuria, nephritis only one of them. Therefore, when the parents found the child has unexplained hematuria, should seek immediate medical attention. Do the relevant checks in accordance with the doctor's recommendation, to be done when necessary routine urine examination to rule out the possibility of renal hematuria.

Three early symptoms of Chronic Kidney Disease: increased urine foam
Increased urine bubble, with a smaller foam-based, and together with the Internet, a long time can not be dispersed, suggesting that children with urine protein, high tension due. Of course, the symptoms of poor accuracy. If you do not have some medical knowledge, parents tend to be overlooked or children. The easiest way is to go to the hospital to check the urine to rule out the possibility of proteinuria. Because of Chronic Kidney Disease is the earliest change urine.

Found early symptoms of Chronic Kidney Disease is not enough, the key is confirmed, then the diagnosis of Chronic Kidney Disease which checks should do more for laboratory tests:? Urinary abnormalities, moderate proteinuria, 24-hour urinary protein excretion at around 2g, often non-selective proteinuria. gross hematuria or microscopic hematuria in urine is one of the changes of Chronic Kidney Disease, with microscopic examination of urine sediment, more than 90 percent of deformed red blood cells hematuria, a small number of homogeneous type erythrocytes hematuria.

Focal segmental glomerulosclerosis with occult onset of proteinuria

FSGS renal medulla
FSGS
Focal segmental glomerulosclerosis (FSGS) nephrotic syndrome in adults the third most common cause (20% to 30%), especially in the black. FSGS is idiopathic, but also related to intravenous drug use, can be found in 20% of non-HIV-positive homosexual persons.

Evidence suggests that a non-immunoglobulin proteins circulating factor-mediated glomerular permeability. Glomerular permeability changes in protein can cause hardening. FSGS renal medulla begin in the near (not easy in biopsy samples). Showed segmental hyalinization, nodular or coarse granular IgM and C3 deposition and diffuse podocyte foot processes disappear. Global sclerosis can occur, leading to glomerular atrophy.

Symptoms, signs and diagnosis

Although sometimes the only signs of asymptomatic non-nephrotic range proteinuria, FSGS patients with nephrotic syndrome usually presents with hematuria, hypertension and renal insufficiency. Typical non-selective proteinuria. IgG levels are often lower. Diagnosis was confirmed by biopsy.

Prognosis and Treatment

Because the treatment is not very effective, and spontaneous remission is rare, so the prognosis is poor. More than 50% of patients with renal failure occurred in 10 years, 20% of end-stage renal failure in two years. The more children, the adult prevalence progress faster. Capillaries disappear (collapsing variation) poor prognosis. Pregnancy can aggravate FSGS.

In 20% to 30% of kidney transplant patients in FSGS recurrence, sometimes within a few hours after transplantation proteinuria appears. Recurrence of FSGS patients, 30% to 50% graft loss; young children, 3 years after the onset of development to mesangial proliferative renal failure and patients with the highest risk.

FSGS induced nephrotic syndrome due to heroin addiction, as early in the disease to stop using heroin can complete remission of nephrotic syndrome.

FSGS sometimes spontaneous regression or corticosteroids effective (such as prednisone 40 ~ 80mg / d8 ~ 12 weeks, followed the next day prednisone 30 ~ 40mg8 ~ 12 weeks). If only mild improvement or recurrence, the plus cyclophosphamide (day 2 ~ 3mg / kg12 week) or cyclosporine (adult daily 5mg / kg or children's daily 6mg / kg16 week) can cause remission. For advanced disease, long-term use of ACE inhibitors tended to reduce proteinuria, and may slow down disease progression. Another option is to merge plasmapheresis tacrolimus immunosuppression. Active treatment can reduce progression to renal failure but increases the risk of serious complications. Anticoagulant and antithrombotic usefulness has not been determined.

What is the treatment of focal segmental glomerulosclerosis


 cause of FSGS

1, treatment
For the treatment of FSGS still controversial past that poor efficacy of this disease, treatment is more difficult, and no mature and effective method of treatment, patients are generally 5 to 10 years into the kidney failure that period. In recent years, a large retrospective study showed a positive result in the removal and treatment of the cause of FSGS, and diuretic, blood pressure and other symptomatic treatment can induce remission of nephrotic syndrome, prevent and delay the progression of the disease. Clinical observations extend FSGS hormone treatments can increase the response rate. At present, for the treatment of patients with FSGS is:

1. glucocorticoid hormone resistance should appear before promptly with prednisone (prednisone) therapy, the dose 0.5 ~ 2.0mg / (kg · d), 6 months. Remission induction dose of at least 60mg / d, 3 months after the reduction to 0.5mg / (kg · d), the average time to complete remission generally get 3 to 4 months. Therefore, the resulting adult FSCS NS after six months of prednisone 1mg / (kg · d) yet remission, it is called hormone resistance. For the elderly, most scholars advocate the next day prednisone 1.0 ~ 1.6mg / (kg · d). For 3 to 5 months. For hormone-dependent resistance and recurrence, intermittent prednisone plus cyclophosphamide pulse therapy can increase the response rate, the total amount of cyclophosphamide should not exceed 12g.

2. cyclosporine (CsA) and cola will be complex (FK506) cyclosporine (CsA) The usual dose 5mg / (kg · d) 6 months of treatment can reduce urinary protein and induce remission, but the reduction or withdrawal often recurrence, therefore, should be long-term use to maintain remission. Renal toxicity due to drug use during serum creatinine should be monitored to adjust the dose accordingly. Coke will resume mechanism with cyclosporine (CsA) is similar to a dose of 0.3mg / (kg · d), can be combined with the hormone. Commonly used in cyclosporine (CsA) therapy or dependent.

3. cytotoxic drugs (cyclophosphamide and chlorambucil oxygen mustard) can be used as two treatments, but its efficacy remains to be confirmed by clinical observation. Banfi, etc. In a retrospective study of patients with FSGS with NS, the patients were divided into three groups, A group was treated with prednisone (prednisone), group B prednisone (prednisone) 1mg / (kg · d ) and chlorambucil, cyclophosphamide or azathioprine in combination with group C with a small dose [0.2 ~ 0.3mg / (kg · d)] prednisone (prednisone) and azathioprine or cyclophosphamide amide MS. Three groups remission rate was 74%, 58% and 38%, respectively. However, the recurrence rate in group A, while the application of cytotoxic drugs recurrence rate of treatment. Tarshish et al reported a prospective study comparing 60 cases of children alone with prednisone (prednisone) and prednisone (prednisone) plus cyclophosphamide 2.5mg / (kg · d) the effect of the two groups did not differ efficient. These data suggest that: the use of hormones or hormone alone with cytotoxic drugs combined, there was no significant difference

Clinical features and treatment of kidney disease FSGS

FSGS
FSGS
FSGS kidney called the Focal segmental glomerulosclerosis, glomerular capillary loop refers to focal segmental sclerosis or hyaline degeneration, no significant cell proliferation of a class glomerular nephropathy.

A common type of FSGS kidney pathology are of primary nephrotic syndrome, 50% of patients with primary nephrotic syndrome pathological type FSGS nephropathy, renal biopsy can clear the disease. The disease mostly occurs in children and adolescents, more men than women, minority FSGS kidney patients before the onset of upper respiratory tract infection or a history of allergic reactions.

In addition, the occurrence of familial FSGS kidney disease may have a genetic predisposition, there atopy in the crowd relatively common kidney disease FSGS, the main clinical manifestations and features:

(1) The first clinical symptoms of nephrotic syndrome, microscopic hematuria is more common, and occasionally gross hematuria, about two-thirds of adult patients with mild persistent hypertension.

(2) Urine examination showed a small number of patients with kidney disease FSGS asymptomatic proteinuria. The vast majority of non-selective proteinuria, but early may have highly or moderately selective.

(3) blood test serum C3 levels were normal, decreased IgG levels.

(4) FSGS patients have kidney proximal tubular dysfunction performance, glomerular filtration rate and effective progressive decrease, upper respiratory tract infection or allergic allows a variety of symptoms.

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